Influenza A virus particles strategically adapt their shape—to become either spheres or larger filaments—to favor their ability to infect cells depending on environmental conditions, according ...

Influenza A virus particles strategically adapt their shape -- to become either spheres or larger filaments -- to favor their ability to infect cells depending on environmental conditions ...

Colorized transmission electron micrograph of influenza A/H3N2 virus particles, isolated from a patient sample and then propagated in cell culture. Influenza A virus particles adapt shape—as ...

For animal influenza viruses to cause pandemics in human populations, their hemagglutinin (HA) proteins must acquire mutations that allow human-to-human transmission. Fortunately, this barrier has ...

To escape this, influenza viruses have developed a special mechanism. They steal the molecular cap from cellular RNA molecules and transfer it to their own RNA. This process is called cap-snatching.

A 1982 study found influenza A remained contagious up to 48 hours on hard plastic or stainless steel, while a 2008 publication found these viruses stayed infectious for up to three days on Swiss ...

Box 1. Current nomenclature for influenza viruses (WHO 1980). The formal nomenclature for the designation of influenza viruses was revised and published by the WHO [43].

In a study of newborn piglets, infection with influenza A was associated with disruptions in the piglets' nasal and gut microbiomes, and with potentially detrimental changes in gene activity in the ...

Trending / Microbiology / Influenza Viruses May Have an Alternate Route Into Cells. JUL 29, 2024 8:08 AM PDT. Share . ... Flu viruses usually use envelope proteins that surround the virus to enter ...

“Since the 1970s, at least, there has been a steady stream of known and emerging viruses, including HIV, Ebola and, of course, influenza comes back every year, sometimes with significant effect.” As ...

More information: Lukas Broich et al, Single influenza A viruses induce nanoscale cellular reprogramming at the virus-cell interface, Nature Communications (2025). DOI: 10.1038/s41467-025-58935-8 ...